Objective To evaluate the systolic function of left ventricular in patients with coronary artery disease (CAD) after cardiac shock wave therapy (CSWT) by two-dimensional longitudinal strain.
Methods A total of 70 patients with CAD were randomly selected, 38 patients were included in the CSWT group, and 32 patients who did not receive CSWT were included in the control group. The patients in the two groups completed dual-nuclides myocardial simultaneous imaging before and 3 months after therapy. The patients in the two groups performed routine echocardiography before, and 3 and 6 months after therapy. Left ventricular ejection fraction (LVEF), left ventricular end-diastolic diameter (LVEDD), left ventricular end-systolic diameter (LVESD), and left atrial diameter (LAD) were measured respectively. The apical long axis (APLAX) view, apical four-chamber (A4C) view, apical two-chamber (A2C) view of left ventricle were collected, and the left ventricular global longitudinal peak systolicstrain (GLS)and GLS-APLAX, GLS-A4C, GLS-A2C were accquired by two-dimensional longitudinal strain.
Results The total score of myocardial perfusion imaging and metabolic imaging in the CSWT group improved 3 months after therapy than before therapy (P < 0.05). There was no significant improvement in the above indexes in the control group after therapy. There was no statistically significant difference in LVEF, LVEDD, LVESD, and LAD in the CSWT group 3 months after therapy. Compared to before therapy, LVEF increased, whereas LVEDD, LVESD, and LAD decreased in the CSWT group 6 months after therapy (P < 0.05). There was no significant difference in LVEF, LVEDD, LVESD, and LAD in the control group before and after therapy. GLS, GLS-APLAX, GLS-A4C, and GLS-A2C all increased in the CSWT group 3 and 6 months after therapy (P < 0.05). There was no significant difference in GLS, GLS-APLAX, GLS-A4C, and GLS-A2C in the control group before and after therapy.
Conclusions CSWT can improve myocardial perfusion and metabolism, two-dimensional longitudinal strain, and the systolic function of the left ventricle, and the improvement of the two-dimensional longitudinal strain may be earlier than LVEF.
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